University of California, Davis BIS 104 Midterm 2 Form A (Contains 33 MCQ and Answers)

EXAM SOLUTIONS > University of California, Davis BIS 104 Midterm 2 Form A (Contains 33 MCQ and Answers)

University of California, Davis BIS 104 Midterm 2 9/06/17 Form A 1. In the Caulobacter bacteria studied in the Shapiro lab the FtsZ gene encodes for a tubulin homologue. Based on your understanding of tubulin activity and the iBiology seminar you watched, you propose that the FtsZ protein binds ________ and surprisingly is required for ______________. A. ATP; DNA replication ...[Show More]

Midterm 2 9/06/17 Form A 1. In the Caulobacter bacteria studied in the Shapiro lab the FtsZ gene encodes for a tubulin homologue. Based on your understanding of tubulin activity and the iBiology seminar you watched, you propose that the FtsZ protein binds ________ and surprisingly is required for ______________. A. ATP; DNA replication B. GTP; contractile ring formation and cytokinesis C. ATP; contractile ring formation and cytokinesis D. GDP; dynamic instability E. GTP; separation of the origin of replication 2. A gradient of the MipZ protein in Caulobacter inhibits Ftsz polymerization at the ends of the cell to ensure proper cell division. An analogous gradient mechanism in animal cells involves a high concentration of __________ on chromosomes that acts to inhibit ________ of microtubules. A. Ran-GTP; catastrophe B. Kinesin 13; rescue C. Kinesin 13; catastrophe D. Ran-GTP; rescue E. Kinesin 5; sliding 3. Blood platelets in the blood stream undergo rapid growth of their actin filaments after activation. Which statement best describes how they begin their rapid actin filament growth? A. There is a high concentration of ARP2/3 to eliminate the nucleation phase. B. Gelsolin actively cleaves actin filaments. C. High concentrations of thymosin keep the G-actin in the ATP form. D. Profilin levels are low, which effectively increases the concentration of free monomeric actin. E. CapZ is bound to the minus end of filaments to keep G-actin concentrations high. 4. A family of patients exhibits severe defects in clotting and platelets that fail to spread after activation. Your research team confirms that gelsolin is active in these patients. Based on your understanding of the filament dynamics necessary to for platelet spreading, you propose that the levels of G-actin are __________ due to the high activity of the __________ protein. A. low; thymosin beta 4 B. high; profilin C. low; CapZ D. high; CapZ E. low; cytochalisn

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